Read e-book online Neurology Volume 72(20) May 19, 2009 PDF

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TLE diagnosis and lateralization of the seizure focus were determined by a comprehensive evaluation including detailed history, video-EEG telemetry, and neuropsychological assessment in all patients. 2 Based on a volumetric assessment that takes into account absolute volume and interhemispheric asymmetry, we classified patients into those with hippocampal atrophy and those with normal hippocampal volume. Eighteen patients refused to undergo surgery at the first evaluation by our epilepsy team. These patients, however, agreed to have repeated MRI scans.

Genotyping using microsatellite markers spanning the SEPT9 gene was also used to identify pedigrees with a previously reported founder haplotype. Ser93Phe) in one HNA pedigree. Sequencing of other known exons in SEPT9 detected no additional disease-associated mutations. A founder haplotype, without defined mutations in SEPT9, was present in seven pedigrees. Conclusions: We provide further evidence that mutation of the SEPT9 gene is the molecular basis of some cases of hereditary neuralgic amyotrophy (HNA).

A randomized, controlled trial of surgery for temporal lobe epilepsy. N Engl J Med 2001;345:311–318. , Wiebe S, French J, et al. Practice parameter: temporal lobe and localized neocortical resections for epilepsy: report of the Quality Standards Subcommittee of the American Academy of Neurology, in association with the American Epilepsy Society and the American Association of Neurological Surgeons. Neurology 2003;60:538– 547. Sperling MR, Feldman H, Kinman J, Liporace JD, O’Connor MJ. Seizure control and mortality in epilepsy.

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Neurology Volume 72(20) May 19, 2009 by AAN Copyright © 2009 by AAN Enterprises, Inc.


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