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By Robert S. Krauss (Eds.)

ISBN-10: 0123744547

ISBN-13: 9780123744548

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Mol. Teratol. 79, 187–210. Haycraft, C. , et al. (2005). Gli2 and Gli3 localize to cilia and require the intraflagellar transport protein polaris for processing and function. PLoS Genet. 1, e53. , et al. (1995). Interaction between undulated and Patch leads to an extreme form of spina bifida in double-mutant mice. Nat. Genet. 11, 60–63. Hol, F. , et al. (1995). A frameshift mutation in the gene for PAX3 in a girl with spina bifida and mild signs of Waardenburg syndrome. J. Med. Genet. 32, 52–56.

Et al. (1995). Interaction between undulated and Patch leads to an extreme form of spina bifida in double-mutant mice. Nat. Genet. 11, 60–63. Hol, F. , et al. (1995). A frameshift mutation in the gene for PAX3 in a girl with spina bifida and mild signs of Waardenburg syndrome. J. Med. Genet. 32, 52–56. Hol, F. , et al. (1996). PAX genes and human neural tube defects: An amino acid substitution in PAX1 in a patient with spina bifida. J. Med. Genet. 33, 655–660. Hoth, C. , et al. (1993). Mutations in the paired domain of the human PAX3 gene cause Klein-Waardenburg syndrome (WS-III) as well as Waardenburg syndrome type I (WS-I).

1988a). A cell-type-specific abnormality of cell proliferation in mutant (curly tail) mouse embryos developing spinal neural tube defects. Development 104, 285–295. Copp, A. , et al. (1988b). Prevention of spinal neural tube defects in the mouse embryo by growth retardation during neurulation. Development 104, 297–303. Copp, A. , et al. (1994). Developmental basis of severe neural tube defects in the loop-tail (Lp) mutant mouse: Use of microsatellite DNA markers to identify embryonic genotype.

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Mouse Models of Developmental Genetic Disease by Robert S. Krauss (Eds.)


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