Download e-book for kindle: Molecular and cellular regulation of adaptation to exercise by Claude Bouchard

By Claude Bouchard

ISBN-10: 0128039914

ISBN-13: 9780128039915

Molecular elements of workout Biology and workout Genomics, the newest quantity within the development in Molecular Biology and Translational technology sequence incorporates a accomplished precis of the proof accrued up to now at the molecular and mobile rules of a number of the diversifications happening in keeping with workout.

Changes within the mobile equipment are defined for a number of tissues and organs by way of signaling pathways, gene expression, and protein abundance. diversifications to acute workout in addition to publicity to common workout also are mentioned and considered.

  • Includes a finished precis of the proof amassed to date at the molecular and mobile law of a few of the diversifications occurring according to exercise
  • Contains contributions from top specialists
  • Informs and updates on all of the most modern advancements within the box of workout biology and workout genomics

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Am J Clin Nutr. 1988;48:240–247. 8. Maughan RJ. Sports Nutrition. Handbook of Sports Medicine and Science. London: Blackwell Science; 2002. 9. McArdle W, Katch FKV. Essentials of exercise physiology. Philadelphia, PA: Lippincott Williams & Wilkins; 2000. 10. van Baak MA. Physical activity and energy balance. Public Health Nutr. 1999;2:335–339. 11. Romijn JA, Coyle EF, Sidossis LS, et al. Regulation of endogenous fat and carbohydrate metabolism in relation to exercise intensity and duration. Am J Physiol.

The best-studied phosphorylation sites are a group of six distinct sites that were identified as substrates for Akt. 78,81,82 Prolonged exercise in humans82–84 and rats,78 as well as AICAR, are also known to cause AS160 PAS phosphorylation. 85 AS160 28 Joram D. Mul et al. 86 These data show that both phosphorylation and calmodulin binding on AS160 are involved in the regulation of exercise-stimulated glucose uptake. These data also suggest that while AS160 may serve as a point of convergence for both insulin- and exercise-dependent signaling in the regulation of glucose uptake, other proteins may be involved in this regulation of glucose uptake.

7 This is primarily accounted for by triglycerides carried within very-low-density lipoproteins (VLDL-TG). TGs are removed from VLDLs by tissue-specific lipoprotein lipases (LPLs). LPL is bound to the capillary endothelium and hydrolyzes TGs, thus liberating FA for uptake into the surrounding tissue (Fig. 1). Tissues with high LPL activity include adipose tissue, skeletal muscle, and heart; however, the regulation of this enzyme is tissue specific. In the fed state, LPL activity is highest in adipose tissue, thereby facilitating storage.

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Molecular and cellular regulation of adaptation to exercise by Claude Bouchard


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