Download e-book for kindle: Etiology and Morphogenesis of Congenital Heart Disease: From by Toshio Nakanishi, Roger R. Markwald, H.Scott Baldwin,

By Toshio Nakanishi, Roger R. Markwald, H.Scott Baldwin, Bradley B. Keller, Deepak Srivastava, Hiroyuki Yamagishi

ISBN-10: 4431546278

ISBN-13: 9784431546276

ISBN-10: 4431546286

ISBN-13: 9784431546283

This quantity makes a speciality of the etiology and morphogenesis of congenital center illnesses. It reports intimately the early improvement and differentiation of the guts, and later morphologic occasions of the cardiovascular approach, masking quite a lot of issues akin to gene capabilities, progress components, transcription components and mobile interactions which are implicated in cardiac morphogenesis and congenital center disorder. This publication additionally provides fresh advances in stem telephone and cellphone sheet tissue engineering applied sciences that have the capability to supply novel in vitro disorder types and to generate regenerative paradigms for cardiac fix and regeneration. this can be definitely the right source for general practitioner scientists and investigators searching for updates on contemporary investigations at the origins of congenital center affliction and capability destiny therapies.

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Download PDF by Toshio Nakanishi, Roger R. Markwald, H.Scott Baldwin,: Etiology and Morphogenesis of Congenital Heart Disease: From

This quantity makes a speciality of the etiology and morphogenesis of congenital middle ailments. It stories intimately the early improvement and differentiation of the guts, and later morphologic occasions of the cardiovascular procedure, protecting quite a lot of themes equivalent to gene features, development elements, transcription components and mobile interactions which are implicated in cardiac morphogenesis and congenital middle disorder.

Additional info for Etiology and Morphogenesis of Congenital Heart Disease: From Gene Function and Cellular Interaction to Morphology

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Moreno-Rodrı´guez, Yukiko Sugi, and Russell A. Norris Abstract In searching for remedial etiologies for congenital heart disease (CHD), we have focused on identifying interactive signaling pathways or “hubs” in which mutations disrupt fundamental cell biological functions in cardiac progenitor cells in a lineage-specific manner. , periostin) and the cytoskeletal regulatory protein, filamin A (FLNA). We discuss them in the context of valve and septal development and the lineages which give origin to their progenitor cells.

Gittenberger de Groot AC, Winter EM, Bartelings MM, et al. The arterial and cardiac epicardium in development, disease and repair. Differentiation. 2012;84:41–53. 7. Zhou B, von Gise GA, Ma Q, et al. Genetic fate mapping demonstrates contribution of epicardium-derived cells to the annulus fibrosis of the mammalian heart. Dev Biol. 2010;338:251–61. 8. Smith CL, Baek ST, Sung CY, et al. Epicardial-derived cell epithelial-to-mesenchymal transition and fate specification require PDGF receptor signaling.

Induction of pluripotent stem cells from adult human fibroblasts by defined factors. Cell. 2007;131(5):861–72. 3. Seki T, et al. Generation of induced pluripotent stem cells from human terminally differentiated circulating T cells. Cell Stem Cell. 2010;7(1):11–4. 4. Kolossov E, et al. Engraftment of engineered ES cell-derived cardiomyocytes but not BM cells restores contractile function to the infarcted myocardium. J Exp Med. 2006;203(10):2315–27. 5. Srivastava D. Making or breaking the heart: from lineage determination to morphogenesis.

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Etiology and Morphogenesis of Congenital Heart Disease: From Gene Function and Cellular Interaction to Morphology by Toshio Nakanishi, Roger R. Markwald, H.Scott Baldwin, Bradley B. Keller, Deepak Srivastava, Hiroyuki Yamagishi


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